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OMA1 mediates OPA1 proteolysis and mitochondrial fragmentation in experimental models of ischemic kidney injury

dc.contributor.authorXiao, Xiao
dc.contributor.authorHu, Yanzhong
dc.contributor.authorMoral Quirós, Pedro 
dc.contributor.authorWei, Qingqing
dc.contributor.authorLópez Otín, Carlos 
dc.contributor.authorDong, Zheng
dc.date.accessioned2015-08-31T10:03:34Z
dc.date.available2015-08-31T10:03:34Z
dc.date.issued2014
dc.identifier.citationAmerican Journal Of Physiology-Renal Physiology, 306(11), (2014); doi:10.1152/ajprenal.00036.2014
dc.identifier.issn1931-857X
dc.identifier.issn1522-1466
dc.identifier.urihttp://hdl.handle.net/10651/32922
dc.description.sponsorshipNational Natural Science Foundation of China [81370791]; National Basic Research Program of China 973 Program [2012CB517600]; National Institutes of Health and Department of Veterans Administration of USA
dc.language.isoeng
dc.relation.ispartofAmerican Journal Of Physiology-Renal Physiology
dc.rights©,
dc.sourceWOS:000337896100007
dc.titleOMA1 mediates OPA1 proteolysis and mitochondrial fragmentation in experimental models of ischemic kidney injury
dc.typejournal article
dc.identifier.local20142108
dc.identifier.doi10.1152/ajprenal.00036.2014
dc.relation.publisherversionhttp://dx.doi.org/10.1152/ajprenal.00036.2014


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