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Cuprizone‐induced neurotoxicity in human neural cell lines is mediated by a reversible mitochondrial dysfunction: Relevance for demyelination models

dc.contributor.authorMartínez Pinilla, Eva 
dc.contributor.authorRubio Sardón, Nuria
dc.contributor.authorVillar Conde, Sandra
dc.contributor.authorNavarro, G.
dc.contributor.authorValle Suárez, Eva María del 
dc.contributor.authorTolivia Fernández, Jorge Luis 
dc.contributor.authorFranco, R.
dc.contributor.authorNavarro Incio, Ana María 
dc.date.accessioned2021-11-09T07:32:45Z
dc.date.available2021-11-09T07:32:45Z
dc.date.issued2021
dc.identifier.citationBrain Sciences, 11(2) (2021); doi:10.3390/brainsci11020272
dc.identifier.issn2076-3425
dc.identifier.urihttp://hdl.handle.net/10651/60839
dc.description.sponsorshipThis research was funded by Fondo de Investigaciones Sanitarias (FIS), which belongs to the Spanish National Instituto de Salud Carlos III though the project (PI15/00601) (Co-funded by European Regional Development Fund and European Social Fund “Investing in your future”).
dc.language.isoeng
dc.relation.ispartofBrain Sciences
dc.rights© 2021 Los autores
dc.rightsCC Reconocimiento 4.0 Internacional
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/
dc.sourceScopus
dc.source.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-85102335823&doi=10.3390%2fbrainsci11020272&partnerID=40&md5=5a652e0ea872ee67b814872909b82017
dc.titleCuprizone‐induced neurotoxicity in human neural cell lines is mediated by a reversible mitochondrial dysfunction: Relevance for demyelination models
dc.typejournal article
dc.identifier.doi10.3390/brainsci11020272
dc.relation.projectIDISCIII/PI15/00601
dc.relation.publisherversionhttp://dx.doi.org/10.3390/brainsci11020272
dc.rights.accessRightsopen access
dc.type.hasVersionVoR


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