Analysis of transcriptomic responses to SARS-CoV-2 reveals plausible defective pathways responsible for increased susceptibility to infection and complications and helps to develop fast-track repositioning of drugs against COVID-19

Andrés Galiana, Enrique Juan de; Fernández Martínez, Juan Luis; Álvarez Machancoses, Óscar.; Bea, Guillermina; Galmarini, C. M.; Kloczkowski, A.

doi:10.1016/j.compbiomed.2022.106029

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Analysis of transcriptomic responses to SARS-CoV-2 reveals plausible defective pathways responsible for increased susceptibility to infection and complications and helps to develop fast-track repositioning of drugs against COVID-19

dc.contributor.author	Andrés Galiana, Enrique Juan de
dc.contributor.author	Fernández Martínez, Juan Luis
dc.contributor.author	Álvarez Machancoses, Óscar.
dc.contributor.author	Bea, Guillermina
dc.contributor.author	Galmarini, C. M.
dc.contributor.author	Kloczkowski, A.
dc.date.accessioned	2023-03-02T09:29:52Z
dc.date.available	2023-03-02T09:29:52Z
dc.date.issued	2022
dc.identifier.citation	Computers in Biology and Medicine, 149 (2022); doi:10.1016/j.compbiomed.2022.106029
dc.identifier.issn	0010-4825
dc.identifier.uri	http://hdl.handle.net/10651/66671
dc.description.sponsorship	We acknowledge financial support from NSF grant DBI 1661391, and NIH grant R01 GM127701 .
dc.language.iso	eng
dc.relation.ispartof	Computers in Biology and Medicine
dc.rights	© 2022 The Authors.
dc.rights	CC Reconocimiento - No Comercial - Sin obra Derivada 4.0 Internacional
dc.rights.uri	http://creativecommons.org/licenses/by-nc-nd/4.0/
dc.source	Scopus
dc.source.uri	https://www.scopus.com/inward/record.uri?eid=2-s2.0-85137168395&doi=10.1016%2fj.compbiomed.2022.106029&partnerID=40&md5=29339a79dfcc063ebde0a0130c47799e
dc.title	Analysis of transcriptomic responses to SARS-CoV-2 reveals plausible defective pathways responsible for increased susceptibility to infection and complications and helps to develop fast-track repositioning of drugs against COVID-19
dc.type	journal article
dc.identifier.doi	10.1016/j.compbiomed.2022.106029
dc.local.notes	OA ATUO22
dc.relation.projectID	NSF/DBI 1661391
dc.relation.projectID	NIH/R01 GM127701
dc.relation.publisherversion	http://dx.doi.org/10.1016/j.compbiomed.2022.106029
dc.rights.accessRights	open access
dc.type.hasVersion	VoR

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